Cross-referencing clinical data
A pairing appears on this page only when a trial gave both ingredients together and measured the result. Bromide has none that clears that bar.
Stitching two separate single-ingredient studies into a pairing is the one thing this engine will not do. When a study of the combination itself holds up at source, it lands here with its citation.
No invented synergy. Where actives were studied on their own rather than together, the record shows each on its own evidence, never a combined effect no trial measured.
Research strength. Research strength says how much work stands behind the combination. It is never a product score.
Independent record. Every finding is cited to a named trial, dated, and never written by the brand.
20 pairings are live across the library today. Checked 20 July 2026.
No study gave these as a pair, so they are not in the card above. But the reason they belong together is settled biochemistry, not a guess, so it is worth knowing.
The sodium iodide symporter transports several halides, and bromide is one of them. High bromide exposure reduces thyroid iodide uptake by direct competition at the transporter. This is textbook halide handling and is the single most relevant interaction for anyone considering bromide intake.
The kidney handles bromide and chloride through the same reabsorption pathways, so raising chloride intake shortens the bromide half life and lowering it lengthens it. This is why saline loading is the established way to clear accumulated bromide. The same is true in the other direction, and a low salt intake slows clearance.
When bromide appears it is nearly always as potassium or sodium bromide, and the counter ion contributes to the daily potassium or sodium load. In formulation terms the salt choice is an electrolyte decision as much as a halide one. It should be counted against total intake, not ignored.
Bromide competes with iodide at uptake, while selenium dependent deiodinases act further downstream in hormone conversion. The two touch the same axis at different steps. Nothing here shows that selenium offsets bromide, and it should not be presented that way.
Algae products are frequently promoted as removing bromide from the body. Bromide is a small freely distributed anion cleared by the kidney, not a heavy metal held by binding agents, so the proposed route does not match how the ion actually behaves. The claim deserves scepticism rather than repetition.
Nothing specific on file for Bromide. Match the label to the daily amount above, and tell your doctor what you take.
Not medical advice. Show the label to your pharmacist.These are the studies our verdict leans on, chosen from the 2 we read for Bromide. The full linked list is below.
12 sources behind our Bromide verdict: peer-reviewed studies and registered clinical trials. Every one links straight to PubMed, the journal, or ClinicalTrials.gov. Read them yourself.
Evidence surfaced via Semantic Scholar (Allen Institute for AI) and ClinicalTrials.gov. Ranked by study type and citation weight, not cherry-picked.
Read this carefully. These are 627,357 voluntary, unverified reactions reported to the FDA (openFDA). The number mostly reflects how popular Bromide is, not how risky it is. A report is not proof Bromide caused anything. It is a signal of what to watch for, nothing more.
Source: openFDA adverse-event reports. Voluntary reporting, not an incidence rate.
FDA Disclaimer: These statements have not been evaluated by the Food and Drug Administration. This information is for educational purposes only and is not intended to diagnose, treat, cure, or prevent any disease. Consult your healthcare provider before starting any supplement regimen.